A link between hypoxia and fetal hemoglobin provides hope for sickle cell disease
Examining how infections can have long-term consequences
12 October 2022Study highlights the importance of earlier contact between mothers and premature babies
12 October 2022A link between hypoxia and fetal hemoglobin provides hope for sickle cell disease
Scientists at St. Jude Children’s Research Hospital have shown how a protein responsible for adapting to low oxygen conditions (hypoxia), causes increased expression of fetal hemoglobin (HbF) in adults. The finding has implications for treating sickle cell disease and beta-thalassemia, serious blood disorders that affect millions of individuals. The research was published today in Nature, with associated code published on GitHub.
