Stopping gene-regulating activity of signaling molecule could aid heart failure prevention

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Stopping gene-regulating activity of signaling molecule could aid heart failure prevention

The human heart works under high demand, constantly pumping oxygen-rich blood through the body. When faced with disease, however, fulfilling this demand can become increasingly difficult and harmful. In the case of chronic high blood pressure—a leading cardiovascular disease in the United States—the heart continuously overexerts, resulting in maladaptive growth and, ultimately, severe dysfunction of the heart muscle itself.

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